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Clinical Endocrinology of Companion Animals by Jacquie Rand, Ellen Behrend, Danielle Gunn-Moore, Michelle

By Jacquie Rand, Ellen Behrend, Danielle Gunn-Moore, Michelle Campbell-Ward

Clinical Endocrinology of better half Animals bargains quick entry to clinically suitable details on dealing with the sufferer with endocrine illness. Written by way of prime specialists in veterinary endocrinology, every one bankruptcy takes an identical constitution to assist within the speedy retrieval of knowledge, delivering info on pathogenesis, signalment, scientific indicators, prognosis, differential prognosis, therapy, analysis, and prevention for a extensive record of endocrine disorders.  Chapters commence with short summaries for speedy reference, then delve into better detail.

With whole insurance of the most typical endocrine illnesses, the booklet contains chapters on stipulations in canine, cats, horses, ferrets, reptiles, and different species.  Clinical Endocrinology of spouse Animals is a hugely sensible source for any veterinarian treating those universal diseases.

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Atypical hypoadrenocorticism: 1. Most patients with atypical hypoadrenocorticism present for gastrointestinal signs, and may have hypocholesterolemia and hypoalbuminemia. Thus, other causes of GI disease with malabsorption, such as inflammatory bowel disease and lymphangiectasia, are often considered concurrently. Additionally, since signs are often exacerbated following a stressful event, dogs with hypoadrenocorticism may be thought to have “stress colitis” or hemorrhagic gastroenteritis. It is imperative that atypical Addison’s disease be considered in these cases so that it is not missed.

E. F. Differential Diagnosis Primary gastrointestinal disease. Toxin ingestion. Renal disease. Pituitary pars intermedia dysfunction (see Chapter 11). Laminitis. Sepsis. VI. A. B. C. Treatment Rest and supportive care. Restore electrolyte balance with appropriate fluid therapy. Glucocorticoid supplementation: 1. Prednisolone sodium succinate (up to 300 mg q 24 h IV or IM) is the initial treatment of choice for hypoadrenocorticism. 2. Low doses of dexamethasone are the second option. 3. Alternatively oral prednisolone (200–400 mg/horse) can be used and may be a consideration for longer term use.

Horses with hypoadrenocorticism have low cortisol concentrations and do not or only minimally respond to ACTH administration: 1. ACTH stimulation test—adult horses: a. Obtain a baseline blood sample in heparinized or plain tubes immediately before ACTH administration. b. Give 1 IU/kg natural ACTH gel intramuscularly (IM) between 8 and 10 am; or 100 IU (1 mg) synthetic ACTH (cosyntropin) intravenously (IV) between 8 am and 12 noon. c. Obtain follow up blood samples 2 and 4 h after ACTH administration.

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